RNF43 p.G659fs 通过 PI3K/AKT/mTOR 信号通路和 HLA-E 上调导致 MSI-high 结直肠癌中 NK 细胞功能障碍
RNF43 p.G659fs leads to natural killer cell dysfunction in MSI-high colorectal cancer through PI3K/AKT/mTOR signaling and HLA-E up-regulation.
CELL INTELLIGENCE · 肿瘤细胞治疗研究
肿瘤细胞治疗研究
靶向肿瘤相关金属蛋白酶并招募 NK 细胞的双特异性抗体——针对口腔癌的多功能治疗策略
英文原题：Bispecific antibodies targeting tumor-associated metalloprotease and recruit NK cells - Multifunctional therapeutic approach against oral cancer.
查看英文原题
Bispecific antibodies targeting tumor-associated metalloprotease and recruit NK cells - Multifunctional therapeutic approach against oral cancer.
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摘要
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论文信息
- 作者
- Kumaresan V、Sivaperumal P、Smiline Girija AS
- 单位
- Department of Molecular Microbiology and Immunology, University of Texas at San Antonio, San Antonio, TX, United States; Department of Microbiology, Saveetha Dental College and Hospitals, Saveetha Institute of Medical & Technical Sciences, Saveetha University, Chennai 600077, Tamil Nadu, India. Electronic address: venkatesh.kumaresan@utsa.edu.United States
- 文献类型
- 读者来信
- 期刊
- Oral oncology2022 Sep
- 原文标识
- PubMed 35809508 · DOI 10.1016/j.oraloncology.2022.106013